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tion accounts for only 144 kbps. The remaining 48 kbps are overhead bits, whose functions are brie y reviewed as follows. The functions covered at the interface include bit timing at 192 kbps to enable the TE and NT to recover information from the aggregate bit stream. This octet timing provides 8-kHz octet timing for the NT and TE to recover the time-division multiplexed channels (i.e., 2B + D multiplexed). Other functions include D-channel access control, power feeding, deactivation, and activation. Interchange circuits are required, of which there is one in either direction of transmission (i.e., to and from the NT); they are used to transfer digital signals across the interface. All of the functions described previously, except for power feeding, are carried out by means of a digitally multiplexed signal. In both directions of transmission the bits are grouped into frames of 48 bits each. However, the frame structures are different in each direction of transmission. A dc balancing bit is periodically inserted to move the signal energy away from 0 Hz.

The reference model of the low rate WPAN mesh is illustrated in Fig. 17.1. In the data plane, the WPAN mesh sublayer resides between the servicespeci c convergence sublayer (SSCS) of IEEE 802.2 logical link control (LLC) and the IEEE 802.15.4 MAC sublayer. The mesh sublayer provides services to the next higher layer via the mesh service access point (mesh SAP). For implementers to add the mesh function to existing low rate WPAN applications with the least effort, the mesh SAP is made very similar to the MAC common part sublayer SAP (MCPS SAP). In the management plane, the mesh sublayer management entity (MHSME) resides between the application management entity (AME) and the MAC layer management entity (MLME). The MHSME also interfaces with the mesh sublayer at the same level. As a reference, the device management entity (DME) that has access and control to all layers is also shown in the gure. However,

Bogartz, R. S. (1994). An introduction to the analysis of variance. Westport, CT: Praeger. Box, J. F. (1978). R. A. Fisher: The life of a scientist. New York: Wiley. Cobb, G. W. (1998). Introduction to design and analysis of experiments. New York: Springer-Verlag. Cochran, W. G., & Cox, G. M. (1957). Experimental designs (2nd ed.). New York: Wiley.

Distributed Database Management Systems by Saeed K. Rahimi and Frank S. Haug Copyright 2010 the IEEE Computer Society

Figure 15.6 Dendrograms for two chosen expertise groups (experts and nonplayers) based on the hierarchical cluster analysis of basic action concepts (BACs) in the tennis serve. The horizontally aligned numbers denote the BACs (for the code, see text); the vertical numbers are the Euclidean distances. For every group, it holds n = 11; p = .05; dcrit = 3.46.

studies have used questionnaires or self-reported subjective arousal, and these ndings suggest that arousal may be a concern for women with diabetes (Schreiner-Engel et al, 1985) Because of a weak correlation between genital and subjective arousal, recent studies have included objective assessments of arousal such as labiothermometry or vaginal plethysmography (Enzlin et al, 1998; Spector et al, 1993), but these ndings are also equivocal (Wincze, Albert, & Bansal, 1993) With respect to the orgasm phase, research ndings are again contradictory and range from signi cantly reduced orgasmic responses in women with diabetes compared to controls (Schreiner-Engel et al, 1987), no decrease (Montenero, Donatoni, & Magi, 1973), or failure to specify orgasmic dif culties as a concern (Jensen, 1981) Rates of dyspareunia, a recurrent or persistent genital pain with sexual intercourse, appear similar to those found in the general population (Spector et al, 1993).

However, Schreiner-Engel et al (1985) found higher rates in women with type 2 diabetes than in controls In women, the role of organic etiologic factors is not as clear or well understood as in men (Cox, Gonder-Frederick, & Saunders, 1991) Although diabetic autonomic neuropathy is believed to be a major cause of organic impotence in men, evidence for a relationship between neuropathy and sexual dysfunction in women is unclear (Spector et al, 1993) Based on the limited research to date, microvascular disease, nephropathy, retinopathy, macrovascular disease, age of onset, duration, and glycemic control tend not to be associated with sexual dysfunction in female diabetes patients (Campbell, Redelman, Borkman, McLay, & Chisholm, 1989; Jensen, 1986) The few studies that included psychosocial factors, such as marital satisfaction (Schreiner-Engel et al, 1985), disease acceptance (Jensen, 1986), and depression (Leedom et al.

, 1991), have found relationships between poorer psychosocial adjustment and sexual functioning in these women In one of the few studies comparing types of diabetes, type 2 diabetes was predictive of sexual dysfunction (Schreiner-Engel et al, 1987), which the authors attribute to the later age of onset of this type of diabetes Treatment of sexual dysfunction in women has also received little recognition in the literature Interventions typically focus on dif culties with arousal and lubrication, with recommendations of diversi cation of sexual behaviors/positions and use of lubricating products Summary The research on sexual dysfunction in diabetes has focused predominantly on men and has supported an organic etiology (autonomic neuropathy) for the primary form of dysfunction,.